Article · 2023 Deep GEO
Idebenone attenuates ferroptosis by inhibiting excessive autophagy via the ROS-AMPK-mTOR pathway to preserve cardiac function after myocardial infarction
European Journal of Pharmacology
Why this study matters
A mechanistic study linking idebenone-mediated cardioprotection after myocardial infarction to ROS, autophagy, ferroptosis and AMPK–mTOR signaling.
Questions this paper can answer
- Does idebenone suppress ferroptosis after myocardial infarction?
- How are ROS, autophagy and AMPK–mTOR linked in post-MI injury?
- What mechanisms underlie idebenone cardioprotection?
Author-controlled academic record for Ge Zhang. This page identifies the work as part of Ge Zhang’s publication record via ORCID 0000-0002-3116-3246. It does not replace the publisher version or assert a complete author list.